MANF Ablation Causes Prolonged Activation of the UPR without Neurodegeneration in the Mouse Midbrain Dopamine System | eNeuro
Molecular mechanism of ER stress‐induced gene expression of tumor necrosis factor‐related apoptosis‐inducing ligand (TRAIL) in macrophages - Huang - 2015 - The FEBS Journal - Wiley Online Library
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
HRC promotes anoikis resistance and metastasis by suppressing endoplasmic reticulum stress in hepatocellular carcinoma
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences
Kinetics of Thapsigargin- Ca2+-ATPase (Sarcoplasmic Reticulum) Interaction Reveals a Two-step Binding Mechanism and Picomolar Inhibition * - Journal of Biological Chemistry
Thapsigargin sensitizes human esophageal cancer to TRAIL-induced apoptosis via AMPK activation | Scientific Reports
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
SEC24A facilitates colocalization and Ca<sup>2</sup><sup>+</sup> flux between the endoplasmic reticulum
Proteomic Signatures in Thapsigargin-Treated Hepatoma Cells | Chemical Research in Toxicology
Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response - Journal of Molecular and Cellular Cardiology
Promising therapeutic effect of thapsigargin nanoparticles on chronic kidney disease through the activation of Nrf2 and FoxO1 | Aging
Knocking out the door to tunicamycin entry | PNAS
Calcium Influx Caused by ER Stress Inducers Enhances Oncolytic Adenovirus Enadenotucirev Replication and Killing through PKCα Activation: Molecular Therapy - Oncolytics
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug
Diabetes Alters Intracellular Calcium Transients in Cardiac Endothelial Cells | PLOS ONE
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text
Cells | Free Full-Text | Calcium Pathways in Human Neutrophils—The Extended Effects of Thapsigargin and ML-9
Silybin regulates P450s activity by attenuating endoplasmic reticulum stress in mouse nonalcoholic fatty liver disease | Acta Pharmacologica Sinica
Luteolin prevents liver from tunicamycin-induced endoplasmic reticulum stress via nuclear factor erythroid 2-related factor 2-dependent sestrin 2 induction - ScienceDirect
What is Thapsigargin? Frequently Asked Questions
Molecules | Free Full-Text | Mipsagargin: The Beginning—Not the End—of Thapsigargin Prodrug-Based Cancer Therapeutics
